Prostate Cancer, Ambrotose and Glyconutrients
A cancer is a group of abnormal cells, known as a tumour, that grows uncontrollably. When cells in some areas of the body replicate without control, the excess tissue that forms is called a tumour or neoplasm. Not all tumours are malignant, some are benign and do not invade and destroy tissue, whereas cancerous tumours do grow and invade surrounding tissue.
Abnormal cell proliferation can be caused by carcinogens, which can be chemical and other environmental factors such as diet and smoking. Stressors to the body such as toxins in food, air, or water cause our body to suffer oxidative stress - production of free radicals. Free radicals (damaged cells) are also produced in response to exercise, emotional or mental stress. Free radicals can cause significant damage to cells. They affect cell components, including proteins, membranes and genetic material. Free radicals can also damage our body's systems and organs. They are one of the chief causes of premature ageing. Free radicals, unless controlled by antioxidants can lead to cancer. Normally if a cell becomes damaged by a carcinogen, it is eliminated by mechanism the body has to deal with defective cells. However, when a cell becomes cancerous, many of these mechanisms are also affected.
The prostate gland sits between a man's bladder and rectum at the base of the pelvis, and is approximately the size of a walnut. The prostate's role is to provide sperm with nutrients and protection. Prostate cancer is the growth of malignant cells in the prostate. The cells reproduce and may spread beyond the prostate gland.
Most prostate cancers grow slowly, but some times the prostate cancer will grow rapidly. This rapid growth is not dependent on the age of the person - it can happen in younger and older men and it can become life threatening. The cancer can spread to the surrounding areas such as the seminal vesicle, lymph nodes in the groin area, the rectum and bones. When cancer spreads to another site, such as bone, the new tumour is still considered to be prostate cancer and not bone cancer
A diet that is high in fruits and vegetables has been shown to offer some protection from many of the negative effects of oxidative stress. High levels of antioxidants and glyconutrients combined provide the best support to deal with free radicals and repair the body.
For all people not just those with cancer, glyconutrients form the basic cellular language of all cells. Cells require adequate amounts of glyconutrients to allow cells to communicate 'correctly'. These nutrients are known to improve the immune system function, organ function and improve basic body physiology. If cells cannot communicate correctly they cannot function correctly or stay healthy. Glyconutrients for cell communication are pivotal no matter what other supplements you decide to take, as they support the body to heal, repair, regenerate, regulate and protect itself
There are four ways glyconutrients in Ambrotose fight cancer. Firstly, they stimulate the immune system's macrophage and immune killer cells to destroy cancer cells. This is the first line of defense. Secondly, glyconutrients increase the production of interferon and other substances to target and destroy cancer cells. Thirdly, glyconutrients activate T-cells so they recognize invaders and destroy them. This is the second line of defense. Lastly, glyconutrients help to regulate when cells die (apoptosis), because it is when this process fails that cancer cells can be replicated.
Cancer begins in cells, the building blocks that make up tissue. Tissues make up the organs of the body. Glyconutrients in Ambrotose work at the cellular level, leading to healthy cells which make healthy tissue, which makes healthy organs, which in turn make healthy bodies.
Disclaimer: The information provided in this medical condition section is strictly educational and not intended as medical advice. For diagnosis and treatment of medical disorders, consult your health care professional.
Copyright 2005 Glyconutrients Reference - Last Updated May 2005
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